464 Oral Treatment Targeting the Unfolded Protein Response Prevents Neurodegeneration and Clinical Disease in Prion-Infected Mice.
http://stm.sciencemag.org/content/5/206/206ra138.abstract
463 Genome-wide association analysis identifies 13 new risk loci for schizophrenia.
http://www.nature.com/ng/journal/v45/n10/abs/ng.2742.html
462 GPR171 is a hypothalamic G protein-coupled receptor for BigLEN, a neuropeptide involved in feeding.
http://www.pnas.org/content/110/40/16211.abstract
461 A specific brain circuit drives the consumption of highly palatable food, even when energy needs are satisfied.
http://www.sciencemag.org/content/341/6153/1517.abstract
460 Erbb4 Deletion from Fast-Spiking Interneurons Causes Schizophrenia-like Phenotypes.
http://www.cell.com/neuron/abstract/S0896-6273(13)00610-7
459 Imaging of Tau Pathology in a Tauopathy Mouse Model and in Alzheimer Patients Compared to Normal Controls.
http://www.cell.com/neuron/abstract/S0896-6273(13)00661-2
458 Suppression of eIF2α kinases alleviates Alzheimer's disease–related plasticity and memory deficits.
http://www.nature.com/neuro/journal/v16/n9/abs/nn.3486.html
457 Top3β is an RNA topoisomerase that works with fragile X syndrome protein to promote synapse formation.
http://www.nature.com/neuro/journal/v16/n9/abs/nn.3479.html
456 By examining the genetic makeup of Northern Finnish sub-isolate populations, Stoll et al. show association between deletion of a topoisomerase III gene (TOP3B) and both schizophrenia and cognitive impairment. They also uncover a mechanism for the concomitant integration of TOP3β with the fragile X protein FMRP into messenger ribonucleoprotein complexes.
http://www.nature.com/neuro/journal/v16/n9/abs/nn.3484.html
455 Germline mitochondrial DNA mutations aggravate ageing and can impair brain development.
http://www.nature.com/nature/journal/v501/n7467/full/nature12474.html
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