| 215  Interleukin-22 alleviates metabolic disorders and restores mucosal immunity in diabetes. http://www.nature.com/nature/journal/v514/n7521/full/nature13564.html
 
 214  Artificial sweeteners induce glucose intolerance by altering the gut microbiota.
 http://www.nature.com/nature/journal/v514/n7521/full/nature13793.html
 
 213  The α2A-adrenergic receptor antagonist yohimbine improves insulin secretion in type 2 diabetics carrying the ADRA2A risk variant.
 http://stm.sciencemag.org/content/6/257/257ra139.abstract
 
 212  Obesity and aging result in elevated levels of central TGF-β, resulting in hypothalamic inflammation and the development of type 2 diabetes.
 http://www.nature.com/nm/journal/v20/n9/abs/nm.3616.html
 
 211  Global genomic and transcriptomic analysis of human pancreatic islets reveals novel genes influencing glucose metabolism.
 http://www.pnas.org/content/111/38/13924.abstract
 
 210  Systemic autophagy insufficiency compromises adaptation to metabolic stress and facilitates progression from obesity to diabetes.
 http://www.nature.com/ncomms/2014/140926/ncomms5934/full/ncomms5934.html
 
 209  Macrophage-inducible C-type lectin underlies obesity-induced adipose tissue fibrosis.
 http://www.nature.com/ncomms/2014/140919/ncomms5982/full/ncomms5982.html
 
 208  Pharmacological fibroblast growth factor 1 (FGF1) normalizes blood glucose in diabetic mice by means of an FGF receptor signalling pathway that is independent of its mitogenic activity.
 http://www.nature.com/nature/journal/v513/n7518/full/nature13540.html
 
 207  Reversible changes in pancreatic islet structure and function produced by elevated blood glucose.
 http://www.nature.com/ncomms/2014/140822/ncomms5639/full/ncomms5639.html
 
 206  MicroRNA-378 controls classical brown fat expansion to counteract obesity.
 http://www.nature.com/ncomms/2014/140822/ncomms5725/full/ncomms5725.html
 
 
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